gene

C2

Entity Detail — Knowledge Graph Node

Understanding Entity Pages

This page aggregates everything SciDEX knows about C2: its mechanistic relationships (Knowledge Graph edges), hypotheses targeting it, analyses mentioning it, and supporting scientific papers. The interactive graph below shows its immediate neighbors. All content is AI-synthesized from peer-reviewed literature.

17Connections
4Hypotheses
1Analyses
17Outgoing
0Incoming
1Experiments
1Debates

Summary

Comprehensive page on C2 (Complement Component 2), a central protein in the classical complement pathway, implicated in Alzheimer's disease, age-related macular degeneration, and systemic lupus erythematosus.

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🧬 Gene Info
Gene SymbolC2
Full NameComplement Component 2
Chromosome6p21.3
Functionencodes a 732-amino acid zymogen that, upon activation, generates the C3 convertase complex essential for complement cascade amplification.
Molecular Weight83 kDa
Amino Acids732 aa
Exons18
UniProt IDP06610
NCBI Gene ID717
Ensembl IDENSG00000166278
OMIM120490
GeneCardsC2
Human Protein AtlasC2
Associated Diseasesautism spectrum disorder
InteractionsMAVS, PI3KC3, SIRT2
KG Connections17 knowledge graph edges
DatabasesGeneCardsHPASTRING
🔮 Predicted Structure: C2 — AlphaFold P06610 Click to expand

AI-predicted structure from AlphaFold | Powered by Mol*

Wiki Pages (21)

Knowledge base pages for this entity

Canonical Page

C2 (Complement Component 2)

gene · 1670 words

SLC2A1 Gene

gene · 2479 words

SLC22A3 — Solute Carrier Family 22 Member 3 (OCT3)

gene · 2439 words

HERC2 Gene

gene · 2395 words

CDC25C — Cell Division Cycle 25C

gene · 2230 words

XRCC2 — X-Ray Repair Cross-Complementing 2

gene · 1993 words

Pathway Diagram

graph TD
    C2["C2"]
    neurodegeneration["neurodegeneration"]
    C2 -->|"implicated_in"| neurodegeneration
    style C2 fill:#006494,stroke:#4fc3f7,stroke-width:2px,color:#e0e0e0
    style neurodegeneration fill:#455a64,stroke:#333,stroke-width:1px,color:#e0e0e0

Outgoing (17)

TargetRelationTypeStr
Huntington's diseaseinhibitsdisease0.60
autism spectrum disorderassociated_withdisease0.60
PI3KC3interacts_withgene0.60
PI3K-AKT-mTOR signalingparticipates_inpathway0.60
autophagy pathwayparticipates_inpathway0.60

Incoming (0)

SourceRelationTypeStr
No incoming edges

Targeting Hypotheses (4)

Hypotheses where this entity is a therapeutic target

HypothesisScoreDiseaseAnalysis
Hippocampal CA3-CA1 synaptic rescue via DHHC2-mediated PSD95 0.861 Alzheimer's disease Circuit-level neural dynamics in neurode
HDAC2 Phospho-Lock Window for Synaptic Gene Silencing 0.643 neurodegeneration What is the optimal therapeutic window d
Oxygen Pressure-Dependent BDNF Cascade: DHHC2/PSD95 Stabiliz 0.580 neurodegeneration View
HDAC2-Specific Repression of PU.1 Pioneer Factor Target Site 0.415 neurodegeneration How does HDAC1/2 deletion specifically e

Mentioning Analyses (1)

Scientific analyses that reference this entity

How do the seven novel ALS genes function in animal models to cause neurodegener

neurodegeneration | 2026-04-13 | 1 hypotheses Top: 0.724

Experiments (1)

Experimental studies targeting or related to this entity

ExperimentTypeDiseaseScoreFeasibilityModelStatusEst. Cost
Synaptic plasticity and protein expression analysis in mPFC exploratory insomnia-anxiety comorbidity 0.850 0.00 mPFC brain slices from PCPA-tr proposed N/A

Related Papers (0)

Scientific publications cited in analyses involving this entity

Title & PMIDAuthorsJournalYearCitations
No papers found

Debates (1)

Multi-agent debates referencing this entity

The authors explicitly state that the effects of these novel genes (MATR3, CHCHD

closed · Rounds: 4 · Score: 0.71 · 2026-04-14

Related Research

Hypotheses and analyses mentioning C2 in their description or question text

No additional research found