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Debates

883 items
IdQuestionQuality ScoreNum Hypotheses GeneratedNum RoundsStatusCreated AtAnalysis Id
sess_SDA-2026-04-12-What is the actual quantitative contribution of FcRn-mediated transcytosis to BB0.9176completed2026-04-12SDA-2026-04-12-gap-debate-20260410-112908-13c403ee
sess_sda-2026-04-01-TDP-43 undergoes liquid-liquid phase separation that becomes pathological. Small0.8476completed2026-04-12sda-2026-04-01-gap-006
sess_SDA-2026-04-03-Does neuroplasticity decline with age?0.5006enrolling2026-04-11
DA-2026-04-11-093252TDP-43 phase separation therapeutics for ALS-FTD0.3406enrolling2026-04-11
DA-2026-04-03-001What are novel CRISPR-based therapies for Huntington's disease?0.5007enrolling2026-04-11
debate-test-enrollmeDoes neuroplasticity decline with age?0.5006enrolling2026-04-11
debate-test-gap-enroTest debate on neuroplasticity mechanisms0.5006enrolling2026-04-11
sess_SDA-2026-04-11-Does reduced Prevotellaceae abundance cause PD pathology or result from it?0.9574completed2026-04-11SDA-2026-04-11-gap-debate-20260410-111558-f9487fea
sess_SDA-2026-04-01-Investigate how lipid raft composition (cholesterol metabolism, sphingolipids) c0.9375completed2026-04-11SDA-2026-04-01-gap-lipid-rafts-2026-04-01
sess_SDA-2026-04-11-TDP-43 phase separation therapeutics for ALS-FTD0.9574completed2026-04-11sda-2026-04-01-gap-006
sess_SDA-2026-04-04-How do alterations in brain lipid metabolism—including gangliosides, phospholipi0.8464completed2026-04-11SDA-2026-04-04-frontier-lipidomics-dcdbc360
sess_SDA-2026-04-04-How do peripheral immune system alterations influence CNS pathology and neurodeg0.5004completed2026-04-11SDA-2026-04-04-frontier-immunomics-e6f97b29
sess_SDA-2026-04-04-How do structural and functional connectivity changes in the human brain connect0.4404completed2026-04-11SDA-2026-04-04-frontier-connectomics-84acb35a
sess_SDA-2026-04-04-What are the critical protein expression changes and post-translational modifica0.5604completed2026-04-11SDA-2026-04-04-frontier-proteomics-1c3dba72
sess_SDA-2026-04-04-Epigenetic reprogramming in aging neurons0.9574completed2026-04-10SDA-2026-04-04-gap-epigenetic-reprog-b685190e
sess_SDA-2026-04-08-The abstract identifies tissue-specific networks that may underlie Mendelian dis0.9576completed2026-04-10SDA-2026-04-08-gap-pubmed-20260406-062222-b5f44522
sess_SDA-2026-04-08-While the abstract mentions identifying subcellular roles of protein interaction0.9577completed2026-04-10SDA-2026-04-08-gap-pubmed-20260406-062222-cc3bcb47
sess_SDA-2026-04-08-The abstract shows that dilncRNAs drive molecular crowding of DDR proteins into 0.9077completed2026-04-10SDA-2026-04-08-gap-pubmed-20260406-062229-35a642ca
sess_SDA-2026-04-08-How do host cell factors influence the conformation and propagation properties o0.9574completed2026-04-10SDA-2026-04-08-gap-pubmed-20260406-062207-b800e5d3
sess_SDA-2026-04-08-While the study shows 53BP1 forms phase-separated foci driven by dilncRNAs, it's0.9577completed2026-04-10SDA-2026-04-08-gap-pubmed-20260406-062229-3ab00c95
sess_gut-brain-adGut-Brain Axis Therapeutics for Alzheimer's Disease?0.9474completed2026-04-10gut-brain-ad
sess_sda-2026-04-01-Why do entorhinal cortex layer II stellate neurons die first in AD? Their unique0.9576completed2026-04-10sda-2026-04-01-gap-004
sess_sda-2026-04-01-PSP and CBD both involve 4R-tau but produce distinct neuropathological patterns 0.9177completed2026-04-10sda-2026-04-01-gap-005
sess_SDA-2026-04-10-How do non-cell autonomous effects of autophagy dysfunction contribute to ALS pa0.9474completed2026-04-10SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9
sess_SDA-2026-04-10-Can circadian interventions reverse microglial priming independent of sleep disr0.9574completed2026-04-10SDA-2026-04-08-gap-debate-20260406-062033-16eccec1
sess_SDA-2026-04-10-Do different priming stimuli require distinct therapeutic approaches or share co0.9374completed2026-04-10SDA-2026-04-08-gap-debate-20260406-062039-f02efa4b
sess_SDA-2026-04-10-What are the minimal structural requirements for HSP70/HSP90 inhibitors to achie0.8674completed2026-04-10SDA-2026-04-09-gap-debate-20260409-201742-5407d57d
sess_SDA-2026-04-10-Do tau-containing vesicles exhibit unique surface glycosylation patterns that di0.9574completed2026-04-10SDA-2026-04-09-gap-debate-20260409-201742-d279750b
sess_SDA-2026-04-10-Which specific post-translational modifications on pathological tau create drugg0.5004completed2026-04-10SDA-2026-04-09-gap-debate-20260409-201742-1e8eb3bd
sess_SDA-2026-04-10-Can nanobodies achieve selective membrane penetration into tau-containing vesicl0.9574completed2026-04-10SDA-2026-04-09-gap-debate-20260409-201742-ca7016f1
sess_SDA-2026-04-10-TREM2 Therapeutic Strategy Post-INVOKE-2?0.9574completed2026-04-10trem2-ad
sess_SDA-2026-04-10-Astrocyte reactivity subtypes in neurodegeneration0.9574completed2026-04-10sda-2026-04-01-gap-007
sess_SDA-2026-04-10-GBA-Synuclein Loop: Therapeutic Strategies for Parkinson's Disease?0.8974completed2026-04-10gba-pd
sess_SDA-2026-04-08-The debate noted clinical failures of TNF-α and IL-6 inhibitors in AD despite th0.7974completed2026-04-10SDA-2026-04-08-gap-debate-20260406-062045-ce866189
sess_SDA-2026-04-08-The debate identified fundamental druggability challenges for these targets due 0.9574completed2026-04-09SDA-2026-04-08-gap-debate-20260406-062052-81a54bfd
sess_SDA-2026-04-08-The highest-ranked hypothesis assumes senescence reversibility through metabolic0.9574completed2026-04-09SDA-2026-04-08-gap-debate-20260406-062101-7751c220
sess_SDA-2026-04-08-The authors identify SPI1 as a potential transcription factor regulating the hub0.9374completed2026-04-09SDA-2026-04-08-gap-pubmed-20260406-062122-bfac06c8
sess_SDA-2026-04-08-The study reports that complement-mediated synaptic elimination produces both co0.8374completed2026-04-09SDA-2026-04-08-gap-pubmed-20260406-062128-34a47c4e
sess_SDA-2026-04-08-While the abstract establishes that intercellular transmission occurs for variou0.9574completed2026-04-09SDA-2026-04-08-gap-pubmed-20260406-062207-5a703c17
sess_SDA-2026-04-08-The study identifies ADCY8 as associated with migratory distance differences and0.9574completed2026-04-09SDA-2026-04-08-gap-pubmed-20260406-062218-580b17ef
sess_SDA-2026-04-08-While APOE4 disrupts microglial metabolism broadly, the debate didn't identify w0.9274completed2026-04-09SDA-2026-04-08-gap-debate-20260406-062033-fecb8755
sess_SDA-2026-04-08-The abstract notes that clinical presentations overlap across different myelopat0.8674completed2026-04-09SDA-2026-04-08-gap-pubmed-20260406-062111-db808ee9
sess_SDA-2026-04-08-The paper describes memory-based migration routes maintained across generations 0.9574completed2026-04-09SDA-2026-04-08-gap-pubmed-20260406-062218-5c7f15f4
sess_astrocyte-subtyAstrocyte Reactivity Subtypes in Neurodegeneration0.9574completed2026-04-09astrocyte-subtypes
sess_sda-2026-04-01-Anti-amyloid antibodies (lecanemab, donanemab) have ~0.1% brain penetrance. Engi0.9174completed2026-04-07sda-2026-04-01-gap-008
sess_sda-2026-04-01-Microglia activate astrocytes via IL-1alpha/TNF/C1q, and reactive astrocytes fee0.9074completed2026-04-07sda-2026-04-01-gap-009
sess_sda-2026-04-01-APOE4 differs from APOE3 by C112R causing domain interaction that alters lipid b0.8974completed2026-04-07sda-2026-04-01-gap-010
sess_sda-2026-04-01-Multiple NDDs converge on autophagy-lysosome dysfunction. Are there universal th0.9074completed2026-04-07sda-2026-04-01-gap-011
sess_sda-2026-04-01-Can speech, gait, retinal imaging, sleep, and smartphone data detect neurodegene0.9574completed2026-04-07sda-2026-04-01-gap-012
sess_sda-2026-04-01-Senolytics targeting p16/p21+ senescent astrocytes and microglia may reduce SASP0.8974completed2026-04-07sda-2026-04-01-gap-013
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