protein

P53779

Entity Detail — Knowledge Graph Node

Understanding Entity Pages

This page aggregates everything SciDEX knows about P53779: its mechanistic relationships (Knowledge Graph edges), hypotheses targeting it, analyses mentioning it, and supporting scientific papers. The interactive graph below shows its immediate neighbors. All content is AI-synthesized from peer-reviewed literature.

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0Analyses
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Summary

JNK3 Protein (c-Jun N-terminal Kinase 3)

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🔬 Protein Info
Gene SymbolP53779
Full NameJNK3 (c-Jun N-terminal Kinase 3)
Protein TypeProtein
FunctionJNK3 is predominantly expressed in neurons and is activated by cellular stress, cytokines, and growth factors.
Primary Expressionneurons and is activated by cellular stress, cytokines, and growth factors
Subcellular Localization| Neuronal nucleus and cytoplasm
Molecular Weight46 kDa
UniProt IDSearch UniProt
GeneCardsP53779
Human Protein AtlasP53779
Associated Diseasesneurodegeneration
DatabasesGeneCardsNCBI GeneHPASTRING
🔮 Predicted Structure: P53779 — AlphaFold SEARCH Click to expand

AI-predicted structure from AlphaFold | Powered by Mol*

Wiki Pages (1)

Knowledge base pages for this entity

Canonical Page

JNK3 Protein (c-Jun N-terminal Kinase 3)

protein · 588 words

Pathway Diagram

graph TD
    P53779["P53779"]
    neurodegeneration["neurodegeneration"]
    P53779 -->|"implicated_in"| neurodegeneration
    style P53779 fill:#4a1a6b,stroke:#4fc3f7,stroke-width:2px,color:#e0e0e0

Outgoing (0)

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Incoming (0)

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Targeting Hypotheses (0)

Hypotheses where this entity is a therapeutic target

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No targeting hypotheses

Mentioning Analyses (0)

Scientific analyses that reference this entity

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Experiments (0)

Experimental studies targeting or related to this entity

ExperimentTypeDiseaseScoreFeasibilityModelStatusEst. Cost
No experiments found

Related Papers (0)

Scientific publications cited in analyses involving this entity

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Debates (0)

Multi-agent debates referencing this entity

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Related Research

Hypotheses and analyses mentioning P53779 in their description or question text

No additional research found