gene

CEBPE

Entity Detail — Knowledge Graph Node

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This page aggregates everything SciDEX knows about CEBPE: its mechanistic relationships (Knowledge Graph edges), hypotheses targeting it, analyses mentioning it, and supporting scientific papers. The interactive graph below shows its immediate neighbors. All content is AI-synthesized from peer-reviewed literature.

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Summary

CEBPE gene - CCAAT/Enhancer Binding Protein Epsilon in neuroinflammation

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🧬 Gene Info

Gene Symbol	CEBPE
Full Name	CCAAT/Enhancer Binding Protein Epsilon
Aliases	CEBPE gene
Chromosome	14q11.2
Protein Type	Transcription Factor
Function	N-terminal transactivation domain (TAD): Responsible for transcriptional activation
Primary Expression	Myeloid cells, microglia, brain
Amino Acids	294 aa
UniProt ID	Q15744
NCBI Gene ID	1053
Ensembl ID	ENSG00000092067
OMIM	605348
GeneCards	CEBPE
Human Protein Atlas	CEBPE
CAAT motif	Canonical C/EBP binding site (ATTGCGCAAT)
Associated Diseases	neurodegeneration
Databases	GeneCards HPA STRING

🔮 Predicted Structure: CEBPE — AlphaFold Q15744 Click to expand

AI-predicted structure from AlphaFold | Powered by Mol*

Wiki Pages (1)

Knowledge base pages for this entity

Canonical Page

CEBPE Gene

gene · 1461 words

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Pathway Diagram

graph TD
    CEBPE["CEBPE"]
    neurodegeneration["neurodegeneration"]
    CEBPE -->|"implicated_in"| neurodegeneration
    style CEBPE fill:#4a1a6b,stroke:#4fc3f7,stroke-width:2px,color:#e0e0e0

Outgoing (0)

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Incoming (0)

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Targeting Hypotheses (0)

Hypotheses where this entity is a therapeutic target

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No targeting hypotheses

Mentioning Analyses (0)

Scientific analyses that reference this entity

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Experiments (0)

Experimental studies targeting or related to this entity

Experiment	Type	Disease	Score	Feasibility	Model	Status	Est. Cost
No experiments found

Related Papers (0)

Scientific publications cited in analyses involving this entity

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Debates (0)

Multi-agent debates referencing this entity

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Related Research

Hypotheses and analyses mentioning CEBPE in their description or question text

No additional research found