entity

cell-type composition shifts

Entity Detail — Knowledge Graph Node

Understanding Entity Pages

This page aggregates everything SciDEX knows about cell-type composition shifts: its mechanistic relationships (Knowledge Graph edges), hypotheses targeting it, analyses mentioning it, and supporting scientific papers. The interactive graph below shows its immediate neighbors. All content is AI-synthesized from peer-reviewed literature.

2Connections
2Hypotheses
2Analyses
0Outgoing
2Incoming
0Experiments
6Debates

No AI portrait yet

Outgoing (0)

TargetRelationTypeStr
No outgoing edges

Incoming (2)

SourceRelationTypeStr
h-gap-456a357b-m2involveshypothesis0.70
h-gap-e7852b55-m2involveshypothesis0.70

Targeting Hypotheses (2)

Hypotheses where this entity is a therapeutic target

HypothesisScoreDiseaseAnalysis
ATAC-seq accessibility separates causal from compensatory st 0.738 neurodegeneration Are DNA methylation changes in neurodege
ATAC-seq accessibility separates causal from compensatory st 0.738 neurodegeneration Are age-related DNA methylation changes

Mentioning Analyses (2)

Scientific analyses that reference this entity

Are age-related DNA methylation changes protective adaptations or pathological d

neurodegeneration | 2026-04-26 | 3 hypotheses Top: 0.750

Are DNA methylation changes in neurodegeneration causal drivers or protective co

neurodegeneration | 2026-04-26 | 3 hypotheses Top: 0.750

Experiments (0)

Experimental studies targeting or related to this entity

ExperimentTypeDiseaseScoreFeasibilityModelStatusEst. Cost
No experiments found

Related Papers (20)

Scientific publications cited in analyses involving this entity

Title & PMIDAuthorsJournalYearCitations
TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS [PMID:33031745] Yu CH, Davidson S, Harapas CR, Hilton JB Cell 2020 2
Chromatin accessibility profiling by ATAC-seq. [PMID:35478247] Grandi, Modi, Kampman, Corces Nature protocols 2022 0
Chromatin accessibility profiling by ATAC-seq. [PMID:35478247] Unknown Nature protocols 2022 0
TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS [PMID:33031745] Yu CH, Davidson S, Harapas CR, Hilton JB Cell 2020 0
DNA Damage, Neurodegeneration, and Synaptic Plasticity. [PMID:27313899] Merlo, Cuchillo-Ibañez, Parlato, Ra Neural plasticity 2018 0
An improved ATAC-seq protocol reduces background and enables interrogation of fr [PMID:28846090] Corces, Trevino, Hamilton, Greenside, Si Nature methods 2017 0
An improved ATAC-seq protocol reduces background and enables interrogation of fr [PMID:28846090] Unknown Nature methods 2017 0
ATAC-seq: A Method for Assaying Chromatin Accessibility Genome-Wide. [PMID:25559105] Buenrostro, Wu, Chang, Greenleaf Current protocols in molecular 2016 0
ATAC-seq: A Method for Assaying Chromatin Accessibility Genome-Wide. [PMID:25559105] Unknown Current protocols in molecular 2016 0
DNA Damage, Neurodegeneration, and Synaptic Plasticity. [PMID:27313899] Unknown Neural Plast 2016 0
Single-cell chromatin accessibility reveals principles of regulatory variation. [PMID:26083756] Unknown Nature 2015 0
Single-cell chromatin accessibility reveals principles of regulatory variation. [PMID:26083756] Buenrostro, Wu, Litzenburger, Ruff, Gonz Nature 2015 0
Human endogenous retrovirus-K contributes to motor neuron disease. [PMID:26424568] Li W, Lee MH, Henderson L, Tyagi R, Bach Science translational medicine 2015 0
Human endogenous retrovirus-K contributes to motor neuron disease. [PMID:26424568] Li W, Lee MH, Henderson L, Tyagi R, Bach Science translational medicine 2015 0
Transposition of native chromatin for fast and sensitive epigenomic profiling of [PMID:24097267] Buenrostro, Giresi, Zaba, Chang, Greenle Nature methods 2014 0
DNA damage and its links to neurodegeneration. [PMID:25033177] Madabhushi, Pan, Tsai Neuron 2014 0
DNA damage and its links to neurodegeneration. [PMID:25033177] Unknown Neuron 2014 0
Transposition of native chromatin for fast and sensitive epigenomic profiling of [PMID:24097267] Unknown Nature methods 2014 0
DNA repair deficiency and neurodegeneration. [PMID:17700067] Unknown Cell cycle (Georgetown, Tex.) 2007 0
DNA repair deficiency and neurodegeneration. [PMID:17700067] Katyal, McKinnon Cell cycle (Georgetown, Tex.) 2007 0

Debates (6)

Multi-agent debates referencing this entity

Formal debate: age-linked CpG drift is the actionable driver in: Are DNA methyla

active · Rounds: 3 · Score: 0.75 · 2026-04-26

Formal debate: ATAC-seq accessibility separates causal from compensatory states

active · Rounds: 3 · Score: 0.75 · 2026-04-26

Formal debate: age-linked CpG drift is the actionable driver in: Are age-related

active · Rounds: 3 · Score: 0.75 · 2026-04-26

Formal debate: ATAC-seq accessibility separates causal from compensatory states

active · Rounds: 3 · Score: 0.75 · 2026-04-26

Are age-related DNA methylation changes protective adaptations or pathological d

closed · Rounds: 4 · Score: 0.78 · 2026-04-26

Are DNA methylation changes in neurodegeneration causal drivers or protective co

closed · Rounds: 4 · Score: 0.78 · 2026-04-26

Related Research

Hypotheses and analyses mentioning cell-type composition shifts in their description or question text

No additional research found