How does FUS loss-of-function in TAZ regulation contribute to ALS/FTD pathogenesis?

Analysis ID: SDA-2026-04-14-gap-pubmed-20260410-184155-2ff305ca | Domain: neurodegeneration | Status: failed | Created: 2026-04-14T10:32:15.956982

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Top Hypotheses (1 total)

#1 ASO-Mediated Exon Skipping to Restore FUS-TAZ Chaperone Axis

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FUS

# ASO-Mediated Exon Skipping to Restore FUS-TAZ Chaperone Axis ## Background and Rationale Fused in Sarcoma (FUS) is a multifunctional DNA/RNA-binding protein belonging to the FET family that plays

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